Placental malaria (PM) is a leading cause of maternal and infant mortality.Although the accumulation of parasitized erythrocytes (PEs) and monocytes within the placenta is thought to contribute to the pathophysiology of PM, the molecular mechanisms underlying PM remain unclear.Based on the hypothesis that excessive complement activation may contrib
Virginie Lemiale, Sandrine Valade, Eric Mariotte Medical Intensive Care Unit, Saint Louis University Hospital, Assistance Publique des Hôpitaux de Paris, Paris, FranceCorrespondence: Eric MariotteMedical Intensive Care Unit, Saint Louis University Hospital, Assistance Publique des Hôpitaux de Paris, 1 Avenue Claude Vellefaux, Paris, 750
Virginie Lemiale, Sandrine Valade, Eric Mariotte Medical Intensive Care Unit, Saint Louis University Hospital, Assistance Publique des Hôpitaux de Paris, Paris, FranceCorrespondence: Eric MariotteMedical Intensive Care Unit, Saint Louis University Hospital, Assistance Publique des Hôpitaux de Paris, 1 Avenue Claude Vellefaux, Paris, 750